Sentence examples for sequestration model from inspiring English sources

The researchers will test a novel sequestration model to explain and understand these results.

We calculated carbon storage using the InVEST carbon storage and sequestration model to estimate aboveground biomass, belowground biomass, soil, and dead organic matter per LULC type.

Using a screening cradle-to-gate LCA and a previously developed and validated CO2 sequestration model for OPC concrete, this paper quantifies and compares estimates of the initial CO2e emissions to the CO2 sequestration potential of several OPC concrete elements at both finite (25 years) and infinite time intervals.

In our previous study, we demonstrated this sequestration model to be feasible, with Sprouty2 showing a higher binding affinity to c-Cbl than EGFR [14].

Unlike the degradation model, the sequestration model proposes that instead of direct import into the pollen tube cytoplasm, both self and non-self S-RNases are directed into a vacuolar compartment sequestered from the cytoplasm.

According to the sequestration model, it is HT-B and not the S-RNase that is degraded (by an as yet unidentified protein) in compatible pollen tubes, whereas in incompatible crosses HT-B remains functional, causing the disruption of the vacuolar compartment and the subsequent release of the sequestered S-RNases into the cytoplasm [16], [19], [29].

This is especially true for carbon sequestration modeling in residential landscapes (e.g., Zirkle et al. 2012).

In the past, LUCC information in large-scale carbon sequestration modeling was not well developed, mainly due to the lack of consistent data describing changes in land use and land cover.

Two models, described as degradation and sequestration models, have recently been proposed to explain how non-self pollen tubes escape the cytotoxic activity of the S-RNase.

Both the recruitment of activator and sequestration models balance X and autosome expression in both sexes.

Two interesting sequestration models have been proposed, based on evidence suggesting that pro-apoptotic factors are mislocalized away from their conventional target sites in infected cells.