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A number of transposons and insertion sequences were present in P. putida genomes indicating their labile nature.
Subsequently, 8808 SSR sequences were found in 7441 unigenes of which 1120 unigenes contained more than one SSR sequence and 507 SSR sequences were present in compound formations.
Where alternative sequences were present in the same these were designated as a mixed genotype infection.
This level of coverage indicated that majority of bacterial sequences were present in the sequenced clones.
Because of amplification difficulties, some data were missing and partial sequences were present in some species.
For example, 695 protein coding sequences were present in Mycobacterium marinum but not in Mycobacterium ulcerans or Mycobacterium tuberculosis [8].
OTU 3 sequences were present in 11.3% of the CD group and in 8.4% of the non-IBD group.
Internal duplications at the DNA level could also be a source of introns if correct splicing sequences were present at the duplicate ends.
No transposon or phage sequences were present in this locus.
Some sequences were present dozens of times among the 581, while other sequences were present only once.
Therefore, some HV1 sequences were present in more than one gene.
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