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Knowledge on the repair efficiencies of DNA damage within such repetitive sequences is therefore crucial for understanding the impact of such domains on genomic integrity.
Amplification of repetitive sequences is therefore frequently used in absolute quantification but problems occur in relative quantification as the number of repetitive sequences is unknown.
A statistical model of CXCR4 sequences is therefore difficult to obtain.
Based on previous results [8], we first hypothesize that genome size is related to fitness, and that the deletion of redundant genomic sequences is therefore an adaptive process.
The number of matches between sequences is therefore related to the lengths of the sequences.
Robust identification of periodic signals in DNA sequences is therefore required to understand nucleosome organisation in genomes.
Similar(53)
These carbonate sequences are therefore described as fringing carbonate platforms that developed laterally and/or above each other.
The GPC and GPD protein sequences are therefore interrupted in the extracellular domain and probably intracellularly degraded.
These sequences are therefore quite stable and would require overcoming a high energy barrier to disassociate some of the existing base pairs.
Protein binding to endogenous and mutated sequences was therefore investigated.
They were consistent with previous data [48] and the sequences were therefore suitable for further analysis.
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