Sentence examples for sequences in normal from inspiring English sources

In comparison, these sequences are only detected in 24.5% (12 out of 49) of all sequences in normal samples.

However, we ruled out the possibility of gene amplification by Southern blotting, which showed comparable level of Bmi-1 genomic sequences in normal and OSCC cells.

We found that for the earliest replicating sequences in normal human fibroblasts, there is a positive correlation with open chromatin, gene and exon density, and active promoters.

This process is promoted by Rad54, a molecular motor protein that not only stabilizes the nucleoprotein filament, but also allows the search for homologous sequences in normal and chromatinized templates [18 20].

TE-derived sRNA species likely regulate the expression/transposition of these mutagenic sequences in normal growth conditions but may also control the transposition events under selective conditions thereby contributing to genome evolution and adaptation.

Identical deletions involving small insertions (3, 4, 6 bp), of unknown origin at the site of the breakpoints, have been previously identified within copy number variants (CNVs) breakpoint-region sequences, in normal population (32, 34).

Finally, we give a criterion for a normal vector space and show that the convergence of a sequence in normal solid vector space has the properties of the convergence in ℝ.

CpG islands in the promoter region of the 47 selected gene targets with CpG islands were bisulfite sequenced in normal mucosal samples from patients without a cancer diagnosis to confirm epigenetic silencing in mature upper aerodigestive tract mucosa (Tables S1 & S2).

DNA methylation of the TPM1 promoter was analyzed by bisulfite sequencing in normal and cancer breast cell lines.

There is a well-balanced remodelling sequence in normal bone: bone is first resorbed by osteoclasts and then osteoblasts form bone at the same site.

Consequently, analysis of global PU.1 DNA binding via ChIP-sequencing in normal haematopoietic progenitors and AML cells in murine, and most importantly patient material, would further clarify the impact of reduced PU.1 activity on target gene regulation.