Sentence examples for sensitivity to pcb from inspiring English sources

PCB 153 exposure was associated with pronounced and long-lasting behavioral changes in SHR/NCrl, suggesting greater behavioral sensitivity to PCB exposure in these animals as compared to WKY/NHsd controls.

Combined, the findings suggest an increased behavioral sensitivity to PCB 153 exposure in the SHR/NCrl as compared to WKY/NHsd controls.

The increased sensitivity to PCB 153 exposure in SHR/NCrl may be caused by exposure effects interacting with the neurodevelopmental time-course through specific effects on cellular mechanisms underlying learning and memory.

We hypothesized that there would be selection in favor of AHR variants that conferred reduced sensitivity to PCBs.

Population or strain differences in gene expression unrelated to sensitivity to PCBs or dioxins have been observed in Fundulus embryos [ 35], Atlantic tomcod embryos [ 61], and strains of rats [ 62, 63].

These results suggest that 6 months of age is not only the time of maximum thymus volume during infancy but may also be the period of greatest sensitivity to PCBs.

Particular focus was given to interaction by sex, given the importance of potential sexual dimorphism in sensitivity to prenatal PCB or MeHg exposures (Castoldi et al. 2008; Cordier et al. 2002; Sagiv et al. 2012a, 2012b).

PCBs may exhibit estrogenic, antiestrogenic, and antiandrogenic effects (Bonefeld-Jørgensen et al. 2001); thus, there may be differences in the sensitivity to these PCB effects between the two sexes because of differences in the natural androgen estrogen balance during critical windows of fetal development.

They do not belong originally to pcb-statechart diagrams.

The lack of interaction we observed between PCBs and p,p′-DDE in increasing sensitivity to induced seizure behavior indicates that PCBs at the levels found in fetal sea lions do not contribute to this response.

First, studies using gene-specific knock-down in zebrafish embryos have shown that AHR2 controls the induction of CYP1A and sensitivity to developmental toxicity of TCDD, PCBs, and PAHs in this species [ 37, 38, 87].