One could argue that higher stress sensitivity drives the inability to experience positive feelings, or that stress sensitivity and positive feelings represent two extremes of one and the same continuum.
It is also worth noting, however, that Δ cpoB is sensitive to a number of other drugs and conditions, including almost all β-lactams to which Δ mrcB and Δ lpoB are sensitive (Nichols et al., 2010 and Figure 2A); these multiple shared sensitivities drive the strong correlation between phenotype profiles of Δ cpoB and Δ mrcB strains.
For instance, individual differences in reward sensitivity, drive, extrinsic versus intrinsic motivational orientation, and trait approach versus avoidance motivation have been found to influence responses to rewarding and aversive stimuli in the ventral striatum, putamen, and caudate (Beaver, Lawrence, Passamonti, & Calder, 2008; Costumero et al., 2013; Linke et al., 2010; Spielberg et al., 2012).
Runoff coefficient sensitivity is driven by elevation (temperature) driven controls on the proportion of precipitation falling as snow vs. rain, combined with decreasing evapotranspiration with increasing elevation.
Taken together, these results indicate that while the principal impact of JNK-deficiency on systemic insulin sensitivity is driven by the parenchymal elements, bone marrow-derived cells also contribute to its total impact, although to a lesser extent.
This would indicate whether the mechanical sensitivity is driven by both C and/ or Ad fibers.
It is likely that much, if not all, of the improvement in insulin sensitivity was driven by improved glucose metabolism within skeletal muscle (32).
This suggests that genome sensitivity is driven by lower gene conservation, involving both gene deletion and movement (duplicate transposition or, to a lesser extent, ancient SSD).
We have shown here that age-related improvements in motion coherence sensitivity are driven by an increase in the effective number of local motion signals that are averaged (i.e., improved global integration).
