Neurological and muscular pathologies, sensitivity deficits (e.g., vestibular, visual), cerebellar syndrome, and many other diseases severely degrade postural control.
Similarly, larger contrast sensitivity deficits were found when stimuli were presented dynamically rather than statically, also suggesting greater M-pathway, than P-pathway, impairment (19).
Although behavioral studies have found contrast sensitivity deficits across spatial frequencies, often thresholds are relatively low (e.g., < 10% contrast; [ 20]), limiting P-pathway involvement.
Finally, decreases in retinal DA metabolism increase the risk of spatiotemporal contrast sensitivity deficits in adult humans and animals (Bodis-Wollner 1990), as observed in monkeys with lifetime Pb exposure (Rice 1998).
(A ) Scotopic (∼3 × 10−4 cd/m) contrast sensitivity deficits in mice lacking RGS proteins (cDKO Cre+) vs cDKO Cre- mice as a function of time of post-tamoxifen administration (*p < 0.05, t-test, n = 4).
In the one case in which this was done on a limited scale [ 17], contrast sensitivity deficits were described as occurring in both the visual hemifield showing no visual loss and the hemifield which did show visual loss.
(E ) Photopic (70 cd/m) contrast sensitivity deficits in mice lacking RGS proteins (cDKO Cre+) vs cDKO Cre- controls as a function of time of post-tamoxifen administration (error bars are SEM; t-test: ***p < 0.001, n = 7).
For high uniformity the effects of crop sensitivity to deficit irrigation are not important and result in only limited (or acceptable) yield reduction and over-irrigation.
Among yield components, number of seeds head−1 showed the highest sensitivity to deficit irrigation followed by number of heads plant−1 while seed weight remained stable.
(5) Rice: rice with full irrigation (because of its high sensitivity to deficit irrigation).
