rad60-SLD2Δ islightlyly sensitive to UV (Figure 1D, Figure S3B) and ionising radiation (Figure S3B).
The upper bands (≈106 kDa) (Figure 1A) were sensitive to neuraminidase (Figure 1B) and PNGase F (Figure 1C) and resistant to Endo H (Figure 1D).
We also observed that swi3-E1, E59, E59 and E68 (Class III), which retained Swi1 Swi3 complex formation, were only sensitive to CPT (Figures 1A and 2A); suggesting that CPT sensitivity is not caused uniquely by a defect of formation of the Swi1 Swi3 complex, and that Swi1 Swi3 possesses at least two separate functions in the preservation of genomic integrity.
They find that human listeners are quite sensitive to such figures.
The P22sTsp was also shown to be completely resistant to trypsin (Figure 2B), but somewhat sensitive to chymotrypsin (Figure 2C) and completely sensitive to pepsin digestion (data not shown).
We found that the ybp2Δ strain was sensitive to benomyl (Figure 1A) and accumulated at the G2/M stage of the cell cycle (Figure 1B), traits common to most kinetochore mutants.
Figure 6 presents the results and shows that the ratios | α ^ K + 1 / α K | stay at low level (< 0.2) and are not sensitive to K'. Figure 6 Means of | α ^ K + 1 / α K | versus given sparsity K' obtained from 10,000 independent Monte-Carlo trials, where α ^ K + 1 denotes the biggest magnitude estimate of spurious sinusoid.
Similar to ChR1 [21], the ChRGR was sensitive to pH (Figure S7).
Indeed, Δtrr1 cells lacking both hba2 and caf5 genes were very sensitive to caffeine (Figure 2D).
Generally, these time delays are not sensitive to ρ (Figure S2 of the Supplemental Data).
The ecsA::intron mutant is also more sensitive to lysostaphin (Figure 1C).
