Sentence examples for sensitive gastric from inspiring English sources
It may help us to identify and target the most sensitive gastric cancer subpopulation for personalized CPT-11 therapy.
First, a cisplatin-resistant cell line BGC823/cis-diamminedichloridoplatinum II) (DDP) was estaBGC823/cis-diamminedichloridoplatinum IIposure of a primary sensitive gastric cancer cell line BGC823.
SULF2 and WRN promoter methylation detection indicates potential predictive biomarkers to identify and target the most sensitive gastric cancer subpopulation for personalized CPT-11 therapy.
Future studies including more genes (such as TOP-I [ 20], PARP [ 21], and Aprataxin APTX) [ 22]) will be carried out to identify and target the most sensitive gastric cancer subpopulation for personalized CPT-11 therapy.
Other complementary mechanisms include the activation of capsaicin-sensitive gastric afferents, stimulation of endogenous prostaglandins and nitric oxide, and opening of K+ATP channels.
CDDP-resistant gastric cancer cells exhibit ABCB1 and CDKN2A gene up-regulation, as compared with CDDP-sensitive gastric cancer cells.
With our PTK profiling technique, we identified two PTKs (eph and hek5) that also responded to RA treatment in RA-sensitive gastric cancer cell lines.
On the other hand, we found that three of four HER2-overexpressing, gefitinib-sensitive gastric cancer cell lines have HER2 gene amplification.
We demonstrated that a new glycoconjugated Pt (II) complex, [PtCl2 (L ], and a new glycoconjugated Pd (II) complex, [PdCl2 (L ], showed significant antitumor effects in CDDP-sensitive gastric cancer and executed their biological effects by inducing apoptosis.
To compare DNA lesions in cisplatin-resistant versus cisplatin-sensitive gastric cancer cells, we determined the phosphorylated histone H2AX (γH2aX), a sensitive surrogate marker of DSB by immunofluorescence staining.
These studies suggest that signal pathways play a positive role in the regulation of P-gp expression In the present study, we assessed p38-MAPK phosphorylation and AP-1 activity in drug-resistant and drug-sensitive gastric cancer cells.
