Once selected, the possibility generators must commit themselves to separating their first step the creation of possibilities from the subsequent steps of testing and selecting.
On the other hand, PCs could benefit from access to the substance information in the ECHA database (at the moment this information is only available for selected governmental authorities), and this possibility should be explored.
Since an appealing hypothesis would be that these peptides are generated by the virus to act as antimicrobial agents that favor viral infectivity in their fish host, we decided to test this possibility by selecting a master peptide and using it to generate substitution variants that may enhance their antimicrobial potential.
Alternatively, the regulation of gene expression in fibrotic glomeruli could be a manifestation (rather than a cause) of fibrogenesis, though we attempted to minimize this possibility by selecting mice for study at an age prior to the appearance of glomerulosclerosis.
However, we control for this possibility by selecting species of the same trophic level, which orientates our prediction towards the hypothesis that large-bodied species are better dispersers and are therefore less sensitive to fragmentation (see Fig. 2Q,R in Ewers and Didham 2006).
To investigate this possibility, we selected one mutation, S33F in MAGEA1, for further study.
To begin to address this possibility, we selected a set of non-candidate genes related to foraging and differentially expressed between two adult behavioral castes of worker honey bees, nurses that perform tasks within the hive and foragers that perform tasks in the field [45].
To test this possibility, we selected horseradish peroxidase (HRP) as a model to find out if Si@poly APBA-PA) can Si@poly APBA-PAlycanrotein from separate.
To explore this possibility, we selected TCGA datasets for these five adenocarcinoma tumor types because of their large patient cohorts and the availability of corresponding clinical data.
To test this possibility, we selected 22 zebrafish and mouse ECRs among the syntenic rodent-bird-fish intronic ECRs from Table 3 and subjected them to an enhancer assay in zebrafish (Additional file 1: Figure S1).
As a model of this possibility, we selected for a population of cells resistant to paclitaxel, and observed the outgrowth of cells with an interferon-inducible gene expression signature originally found in only a minor population of paclitaxel-insensitive cells in the parental cell line.
