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In the B regime, the product sδDGB was determined (s being the solute GB segregation factor, δ the GB width, DGB the GB diffusion coefficient), while in the C-regime DGB values were measured directly.
In the B regime the triple product P=s·δ·Dgb (where s is the segregation factor and δ the GB width) was found to obey the Arrhenius law P=6.6×10−12×exp (−102.8 kJ mol−1/RT) mol−1/RT
Knowing the segregation factor, the experimental data on Ag diffusion along both nanocrystalline and inter-agglomerate interfaces in the nanomaterial were systematically analyzed in dependence on the different kinetic regimes.
The results of various multi-scale GBSE models with and without traps (including the effects of microstructure, intergranular precipitate phases and GB thickness) are compared and discussed, and the effects of microstructural parameters such as hydrogen segregation factor and GND trapping density on hydrogen diffusion are investigated.
The independent measurements in Harrison's C and B kinetic regimes resulted in direct data of the GB diffusivity Dgb and of the so-called triple product P = s · δ · Dgb (s and δ are the segregation factor and the diffusional GB width, respectively).
In the B-type diffusion regime, the triple product P=sδDgb (s is the segregation factor and δ the GB width) was found to follow the Arrhenius law P=(1.4+0.6−0.4 ·10−15·exp{−[(69.1.4+0.6−0.4 ·10−15·exp{s−1.4+0.6−0.4 ·10−15·exp{
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Combining the obtained sδDGB and DGB values and assuming δ=0.5 nm, the GB segregation factors and segregation enthalpies of Ni (HsNi=−39.7 kJ/mol) and Se (HsSe=−21.4 kJ/mol) in Ag were evaluated.
Perhaps not surprisingly, mps3 SUN domain mutants exhibited negative genetic interactions with chromosome segregation factors and components of the spindle checkpoint.
Evidence from this study suggests that a novel stimulus for MHC isoform switching in the adult heart is genetic segregation of factors associated with low intrinsic aerobic capacity.
Infertility is a common characteristic of hybrid yeast strains due to the incompatibility of genes coming from the parental genomes, gross chromosomal rearrangements and abnormal gene segregation, among other factors.
The study was the first to look at segregation as a factor in lung cancer mortality.
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