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(D) Quantification of the mean vessel density in tumor sections demonstrates a highly significant decrease in vessel density in the therapy group, **P < 0.001.
The analysis of 3888 microspheres distributed in 240 sections demonstrates that the increasing non-uniformity with increasing mean absorbed dose per biopsy is caused by the aggregation of large clusters in small arteries.
(C) TUNEL staining of corresponding tumor sections demonstrates a higher number of apoptotic cells in the SU11248-treated tumors, TUNEL-positive cells in red, counterstaining of vessels by collagen IV (green), and nuclei stained by DAPI (blue), bar = 100 μm; (D) Quantification confirms a significantly higher ratio of the TUNEL-positive area fraction in treated tumors, **P < 0.001.
Histological examination of tissue sections demonstrates a diffuse pattern of myelin loss throughout the corpus callosum as well as the fimbriae, the external capsule and the deep white matter.
The analysis of the previous sections demonstrates that transport of G-actin in both Case-0 and Case-1 of our model is dominated by simple diffusion and that transport via cytosolic flow is negligible.
(G ) AChE histochemistry on fresh frozen brain sections demonstrates a significant reduction of striatal AChE in Dlx-CKO mice (t-test; t(22) = 5.16; p < 0.0001).
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Other sections demonstrate national pride in industrial and transportation achievements.
Histological sections demonstrated an enlarged globe with focal scleral thinning.
This chapter describes the behavior of these sections, demonstrating their structural advantages using testing and finite element method modeling.
The results of the previous sections demonstrated the inferior performance of 3D-based approaches in comparison with 2D-based methods.
The histological sections demonstrated the marked difference in the bacterial virulence between S. aureus and S. epidermidis strains.
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