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Some are pathogens of humans (V. cholerae and V. vulnificus), animal symbionts (e.g. V. fischeri in the squid) or deep sea bacteria (Photobacterium).
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Blast analyses using protein sequence showed that all three subsets of CDSnc of S. halifaxensis and S. sediminis were best matched in the cold deep sea bacterium P. profundum, with the exception of S. sediminis CDS2-sw that was best matched in cold-adapted marine bacterium C. psychrerythraea.
Another clade, which harbored C. subterminale was associated quite closely (BV 703) with Clostridium tunisiense, and red sea bacterium on one clade, Clostridium sulfidigenes, C. thiosulfatireducens and bacterium isolates (BV 852-1000) on a second clade and yet another branch carried swine manure bacterium (BV 961).
These organisms, Mariprofundus ferrooxydans (a deep sea bacterium collected near Hawaii), candidate division TM7 (collected from a subgingival plaque in the human mouth), and marine gamma-proteobacterium (collected in the coastal Pacific Ocean at 10 m depth) were detected with low log-odds scores on numerous experiments using different samples.
However, our understanding of the roles of SOS pathway in deep-sea bacteria is limited.
However, our understanding of the roles of CRP in deep-sea bacteria is rather limited.
Pressure is thought to have a significant influence in deep-sea stratification, as piezophilic (barophilic) species have been isolated [18], and specific adaptations such as pressure regulated operons are present in some deep-sea bacteria [19], [20].
This study opens up avenues for identifying new DNA damaging compounds from deep-sea bacteria.
However, the role of deep-sea bacteria in SON degradation and cycling has never been analyzed at a genomic level.
Many deep-sea bacteria produce exopolysaccharides that help them survive in the extreme deep-sea environment [ 30].
Through phosphorylation relay between regulator proteins, some transcription promoters can be activated to regulate hydrostatic pressure in deep-sea bacteria.
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