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After reaching a PaCO2 equilibrium (variation < 5% in three consecutive arterial blood samples), we considered that the stabilization phase was finished.
Additional comments need to be made regarding the samples we considered in the analysis.
Table V shows the different methods (i.e. parameter restrictions and availability of control samples) we considered.
For comparison with gene expression measured in whole-tissue samples, we considered the set of probes that were present on the two array chips.
As the ratios of unmapped reads were almost the same in the control and infected samples, we considered that few reads were derived from fungal transcripts.
As potential patient samples we considered hospitalized children and adults of every age and indication, whose treatment involved the management strategy "clinical pathways".
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In generating a set of samples, we consider only the variations of parameter values in the conditional distributions (Tables 2 (right group) and 3 (upper right).
By combining single probe data across multiple samples, we consider the entire population of probe expression values (gene values) as derived from a single distribution.
Inferring LD from Using Nonrandom Samples: we consider two biallelic loci M (marker locus) and A (disease locus).
However, the size of the sample we considered may have affected the West African results at both regional and national levels.
We then performed 10 simulations of the germ-grain model with parameters (underline{p}) and for each simulated sample we considered one section out of two, like in the real material.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com