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By analyzing luminescence spectra of the low-loading samples, we conclude that the type A catalyst contains monomeric NbO4 tetrahedra and the type E catalyst oligomeric NbO4 tetrahedra.
Overall, given the lack of consistent replication across all our screened sample sets and the lack of SNP associations in the largest sample set (i.e. combined analysis of all samples), we conclude that MEFV is unlikely to contribute to UC susceptibility.
On the basis of previous serologic investigations that found anti-OPV antibodies in lynx in Sweden (25 ) and our detection of OPV-specific DNA in lynx tissue samples, we conclude that the Eurasian lynx in Sweden are infected with OPV.
Based upon our molecular analysis of primary cutaneous (BCC, SCC, PCM) and MM samples, we conclude that PCM and MM have distinct gene expression profiles that may be useful for tumor tissue classification.
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By comparing the effective lifetime of SiNWs/Al2O3 and c-Si/Al2O3 samples, we concluded that the thermal annealing conditions, especially temperature, significantly affect the state density at the SiNW/Al2O3 interface.
In our current study, considering the relatively elevated levels of TGF- β3 in PDR samples, we concluded that TGF- β3 was linked to PDR, which is characterized by retinal neovascularization.
Because our observations do not differ from those published previously for other cultivated and wild rice samples, we concluded that abscission layer traits are robust under our growth conditions, and we did not sample additional time points of abscission layer development.
As, consequently, somatic mutations with substitution of amino acids or frameshift were not seen in these samples, we concluded that ATBF1-A mRNA levels may be regulated at the transcriptional stage, but are not regulated by genetic mechanisms, deletions (LOH), or mutations in breast cancer.
Despite the variability between patient samples, we concluded from these data that sorafenib was directly cytotoxic to human primary sarcomas ex vivo with a corresponding increase in ALDHbright cells, and our ex vivo results with pazopanib and regorafenib correlated with our results in vitro.
The tests for deviations from linkage disequilibrium were significant in 3 of 90 comparisons at P < 0.01; however, because the significant linkage tests involved different pairs of loci in different samples, we concluded that they were more likely effects of type I errors than physical linkage between loci.
Based on our wide taxon sampling, we conclude that Tas1r1 is lost or pseudogenized in most, if not all, bat lineages.
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