Besides the overall MSD over all test samples, we assessed two additional MSD measures for both small trees (MSD1) and large trees (MSD2).
To circumvent this problem, in a subset of samples, we assessed the concordance between the extents of methylation in the promoter-CGI(s) of each gene (using UCWBC DNA samples) with its respective transcript expression levels in RNA samples isolated from matched FPT samples.
To understand how FLT3 receptor and NPM1 mutational status relate to intracellular biological pathways in diagnostic AML samples, we assessed IL-27 induced Jak/Stat signaling and FLT3L induced PI3K and Raf/Ras/MAPK signaling responses in FLT3 receptor and NPM1 molecular defined subgroups.
In 11 control samples, we assessed the Ct values of ABL1, B2M, GUSB, and JAK2 genes.
This may be the reason why this pathway is universally aberrant in all the LUAD samples we assessed.
Among the 58 paired samples, we assessed the changes in fluorochemicals over time using the Wilcoxon signed-rank test.
Because we had detailed clinical laboratory data from our KD subjects concurrent with the whole blood RNA samples, we assessed whether cell subtype numbers affected gene ontology.
We hypothesized that TBB is rapidly metabolized to TBBA; thus, it is likely that the spot urine samples we assessed are reflective of recent exposure (< 1 day previous).
Using all available Fairview samples, we assessed the occurrence of full and half-sibling relatedness within galleries, and among galleries within sections, sections within trees and among trees.
On eight matched, randomly selected HMD and LMD samples, we assessed the collagen structure using SHG imaging coupled with GLCM texture analysis.
