The signals from the five spike mRNAs were used as a dose-response standard curve for each sample, enabling us to convert all the signals measured to copy numbers per cell in an expression profile-independent manner.
Having two barcodes in the same PCR sample enabled us to detect template-switched isoforms in the final library.
Despite the relatively short follow-up, the inclusion of a large sample enabled us to demonstrate the poor prognostic role of HER2 ECD level.
Recruitment of a purposive sample of participants from a large sample enabled us to collect data from families with a range of different levels of PA and SV, from different deprivation groups.
Deep sequencing of six samples per treatment produced an average of more than 10 million reads per sample, enabling us to profile the miRNA expression with high depth and identify miRNAs significantly affected by the diets with high confidence.
The multiple loss experiences reported by our study sample enabled us to examine a possible "dose-response-effect" as has been documented in the trauma literature for the development of PTSD [for example 3].
The large size and longitudinal nature of our strain sample enabled us to obtain extensive information about the level of emm allelic variation and the rate of emergence of new emm alleles in Ontario.
MS analysis indicated a single chemical species, and backbone resonance assignment experiments on a U-C,N-labelled sample enabled us to establish the sequential connectivities for these minor cross-peaks, as illustrated for the residue sequence Thr His.
Although many of the woman with advanced or metastatic cancer would have been deceased by the time of our data collection and so we would likely have been able to obtain their data without consent, we would argue that having the benefit of the whole sample enabled us to produce these comparisons with some confidence.
Using the same samples enables us to correlate datasets collected using different analytical techniques.
