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Routine coagulation markers were measured.
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Clinical data collected for each patient included lactate dehydrogenase (LDH) and β2-microglobulin (β2-MG) levels, immunoglobulin (IG), blood biochemical functions, liver function, blood routine, coagulation functions, tumor markers, ferritin, bone marrow smear, and chemotherapy regimens.
Calculation of the score requires only routine coagulation tests: platelets count, assay of a fibrin-related marker, prothrombin time and fibrinogen.
Perhaps the routine inclusion of additional tests for patients with suspected severe CAP such as PCT and coagulation markers (for example, D-dimer, prothrombin fragment 1.2 or thrombin-antithrombin complexes) may identify patients who require additional supportive care to address these disorders, either through admission to ICU or through initiation of additional treatment.
The relationship between routine coagulation parameters and platelet count has not been explored.
There is no correlation between routine coagulation values and platelets at admission.
Thus thromboprophylaxis would often be used inadequately if the indication were based on coagulation markers.
Routine coagulation tests evidence a prolongation of both prothrombin time (PT) and activated partial thromboplastin time (aPTT).
Laboratory analysis including routine coagulation parameters was conducted on on a daily basis during the ICU stay.
Interventions: Blood samples for thromboelastography and routine coagulation tests were collected before induction of anesthesia and at 10 and 60 minutes after protamine reversal of heparin.
Routine coagulation of all nonbleeding visible vessels (NBVVs) in post-ESD ulcers is currently performed as standard practice, but it cannot eliminate bleeding.
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