Solution phase experiments were also conducted to help shed light on whether reactive oxygen species (ROS) contributed to the photochemistry of As III) in the presence of birnessite.
Furthermore, our results indicated that dysregulation of ROS contributed to the oligomerization and translocation of Drp1, resulting in mitochondrial fragmentation in rhNPC.
It has been well documented that ROS contributed to the induction of genomic instability and carcinogenesis.
PPD-induced ROS contributed to the survival of colorectal cancer cells via activation of the NF-κB pathway.
As radiation-induced secondary factors, ROS contributed to the radiation-induced DNA double-strand breaks (Riley, 1994) and could concurrently trigger the release of cyt- c from mitochondria in the irradiated cells (Chen et al, 2009; Ogura et al, 2009).
Furthermore, another study that evaluated the role of ROS in the interactive toxicity of carbide-cobalt mixtures found no evidence that production of ROS contributed to the toxicity of WC-Co in macrophages [ 32].
