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Following a 3 h treatment with different-sized ZnO NPs (4, 13 and 20 nm), a size-dependent induction of ROS was observed at both NP concentrations, with 4-nm-sized NPs consistently inducing higher levels of ROS compared to 13- or 20-nm-sized NPs (Fig. 8).
Consistent results were observed in N20 cells that exhibited reduced level of ROS compared to control (Fig. 4d).
In RC0.1 cells, CPT treatment induced a higher production of ROS compared to parental DU145 and H460 cells (figure 2 B).
We found that neutrophils from the patients produced significantly lower levels of ROS compared to their individually matched controls (with a mean reduction of 30% ±17 Fig. 3E F).
Additionally, F3 showed higher inhibition of ROS compared to the crude seed extract (p < 0.05).
In the Low ROS compared to the control group, 12 DEPs involved in reproduction and fertilization were under-expressed.
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We found that pri-mir-92a expression in the posterior T2 femurs is expanded in e, wo, ro compared to Oregon-R.
The interpretations by the ROs were compared to the likelihood ratios (LRs) obtained from a probabilistic model that allows a calculation of LRs to assist the interpretation of LT DNA evidence and both were compared to the true composition of the designed mixtures.
No brain regions were more activated in RO as compared to thin individuals in the eucaloric state (EU RO>Thin:H>O).
Conversely, in apical synapses, hippocampal slices from the left exhibited a greater sensitivity to Ro 25-6981 compared to the right (right, 76±4% of control, n = 7 from 7 animals; left, 38±4% of control, n = 7 from 7 animals; P<0.01)(Apical, Figure 1C).
In agreement with the Cin-sensitivity results, ∼4 times as many FAD and SAD cells were stained with FL-SA-Ro as compared to controls.
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