Sentence examples for rodent model was from inspiring English sources

Using a similar experimental paradigm, we have previously demonstrated that prenatal undernutrition in a rodent model was associated with a prepubertal increase in food intake [29] and alterations in epigenetic regulation of and mRNA levels of hepatic genes to favour lipogenesis [42].

Since this rodent model was only recently introduced, there are no extensive data pertaining to its histological and genetic markers.

A similar rodent model was utilized by Moriyama et al. who observed improved tibial implant integration dose-dependently after 7 14 days of local fluvastatin-release from a PGA-coating [ 26].

The CNS-1 rodent model was then used to identify ADAMTS5 (a disintegrin and metalloproteinase with thrombospondin motifs 5) as the MMP required for proteolytic cleavage of BAHAB/brevin (Viapiano et al., 2008).

The rodent model was first introduced in the 1980s and is most widely used nowadays because of its easy accessibility, low cost, and shorter duration to obtain clinically relevant observations.

The overexpression of wild-type TDP-43 in the nuclei in a transgenic rodent model was sufficient to be toxic to spinal motoneurons (Li et al., 2010; Shan et al., 2010; Wils et al., 2010; Xu et al., 2010), which is consistent with our observations in the rat model.

The MCD-fed rodent model is widely used as a NASH model because of its similarity with human NASH pathology.

The advantage of using this particular model is that the estimated g force, rotational measures and HIC15 values for our rodent model are sufficient to produce both behaviorial and histopathological changes (gliosis) indicative of concussion (without fracture to the calvarium).

The estimated g force and HIC15 values for our rodent model are the sufficient to produce behavior and histological changes (astrocytosis) indicative of mild traumatic brain injury or concussion without fracture to the calvarium.

Using these considerations, analysis of ribozyme gene therapy for an autosomal dominant RP-like disease in a rodent model is outlined emphasizing the desirability of combining biochemical, morphological and electrophysiological measures of therapy.

Child neglect is the most common form of child maltreatment, yet the biological basis of maternal neglect is poorly understood and a rodent model is lacking.