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Relative to geldings receiving oil, all geldings receiving estradiol injections had a rise (P <.05) in estradiol concentrations lasting at least 12 days.
In heifers treated with OIF on Day 6, the dominant follicle diameter profile tended to be smaller than in controls, and was associated with a rise (P < 0.05) in plasma FSH concentrations.
All doses of substance P caused an immediate rise (P <.01) in plasma prolactin concentrations, with the three highest doses producing similar responses, and the 62 μg dose producing a minimal response (P <.05).
In males, univariate regression analysis revealed strong associations between walking speed and a number of predictors including age (p < 0.01), muscle area (p < 0.01), intermuscular adipose tissue area (p < 0.01), and rate of EMG rise (p < 0.001).
Hypo-salinity caused a significant rise (P < 0.05) in the mRNA expression of CHH and gill Na+/K+-ATPase activity at 12 h and 24 h respectively, then the CHH mRNA expression declining by 24 h, and returned to control group level by 48 h, and the Na+/K+-ATPase activity tended to be stable after 72 h, and higher than that of control.
In addition, HLA-B*48 (3.6 fold rise, P = 0.304 in DF; 3.1 fold rise, P = 0.333 in DHF) allele was increased in both patient groups.
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Over time, cardiac output significantly rose (P < 0.05) and (P < 0.05).
These patients also had significantly higher renal indices including estimate glomerular filtration rate changes (p = 0.041) and postoperative urea level rises (p = 0.0286).
Among NCCM risk factors, fat mass increased with increasing quintiles of Cac (p value = 0.001); LDL-cholesterol increased (p value<0.001) whereas LDL size decreased as Cac quintiles rose (p value = 0.001).
Following NCT only two patients had significant rises (P < 0.05) in tumour median pO2.
Administration of TQ significantly modulated these alterations and the level of GSH rose (P < 0.05) as shown in Table 2.
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