Survivorships were homogenous among the four reversion lines (P=0.1198, Kruskal-Wallis test).
Survivorships of the four reversion lines were homogenous (P=0.1962, Kruskal-Wallis test).
Overall, the nature of the differences among the reversion lines remains obscure.
Second, these authors created reversion lines, that is, lines that evolved first on a novel host and subsequently evolved on the ancestral host again.
Under the null hypothesis that reversion lines did not experience any loss of fitness on the novel host after reversion, the slope of the curve linking performance at the time of reversion to performance at the time of the assay, should not significantly differ between control lines and reversion lines.
A significantly decreased lifespan was detected in mated mutant females compared to control females and females of the four reversion lines (Table 1, Figure 3A).
A significantly increased lifespan was detected in unmated mutant females compared to control females and to females of reversion lines rev1, rev4 and rev5 (Table 1, Figure 2A).
We assessed the effect of the stc KG01230 mutation on lifespan relative to control and reversion lines in unmated and mated females and males.
Consequently, survival curves were not homogenous among control females and females of all reversion lines (P=0.0003, Kruskal-Wallis test), though mean and median lifespans of control females and females of all reversion lines were almost identical and varied between 53 and 55 days (Table 1).
Survivorships of all reversion lines were not different from survivorship of the control line (P=0.2542, P=0.3366, P=0.6245, P=0.4784, respectively, Kruskal-Wallis test), consequently, survivorships were homogenous among all five lines (P=0.2110, Kruskal-Wallis test).
