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Our study revealed lacking protection by PON2 against receptor-triggered signaling.
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These complementary experiments validated some of the predictions but also revealed lacks in the generic network.
An ~550 base-pair (bp) amplicon was observed, which sequencing revealed lacked exons 20 and 21 of RBM6 and exon 1 of RBM5.
Characterization of the patient's B cells revealed lack of surface CD19 although both of her CD19 alleles were normal.
FT-IR revealed lack of significant interactions between NPX and HPMC.
However, pair wise comparisons (Tukey tests) revealed lack of significant differences within factors for certain levels.
DSC and FT-IR studies revealed lack of significant interactions between CIN and PVA.
Fracture surface examination and μCT scans revealed lack of fusion defects, disbonded regions, and random/isolated porosity for the as-deposited samples tested in this study.
Host-pathogen RNA-sequencing revealed lack of a pyrazinamide exposure transcript signature in intracellular bacteria or of phagolysosome acidification on pH imaging.
Two locus interaction analyses revealed lack of significant interaction (Table S6).
The interviews of half of patients revealed lack of ongoing assessment.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com