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This paper presents the application and the validation of the SPH numerical model SPHyCE, based on a standard Smoothed Particle Hydrodynamics formulation, for modeling an onshore oscillating water chamber, comparing results of amplification factor and phase lag with those obtained by a mesh-based RANS model.
The results of amplification were analyzed by the comparative threshold cycle method, also known as the 2-ΔΔCt method [ 60].
The results of amplification studies using homozygous substrates mixed in different ratios were ambiguous and were not able to clearly identify a cause (data not shown).
Results of amplification reactions were validated by expected findings for water blanks, negative controls, and positive controls derived from viral culture lysates (adjusted to concentrations representative of patient specimens) included in each PCR reaction plate.
Both the results of amplification and IHC can be affected by subcellular modifications (i.e. crosslinking between proteins and/or nucleic acids that provides a strong steric hindrance creating an intricate physical barrier) induced by these treatments [ 15].
The final results of amplification of the target products indicated no obvious differences between the two primer types, i.e. they presented similar sizes of products on the agarose gels (data not shown).
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The result of amplification displayed that only the fresh and the cooked meat samples from the Chinese alligator could be amplified with two bands.
This paper reports the PCR vessel structure, chip package problems and solutions, its thermal uniformity analysis, thermal cycling properties and the result of amplification.
If our assumptions and analysis hold, the first node should receive a constant valued signal at baseband, showing no frequency offset as a result of amplification and retransmission by the relay.
Increased secondary mechanical sensitivity has been shown to be the result of amplification generated, in part, by sensitized dorsal horn neurons [49].
However, we have focused our analysis upon a core of 30 genes that are overexpressed either as a result of amplification of chromosome 17 (in ER+) or deletion of chromosome 8 (in ER−).
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