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Resolving these possibilities ultimately requires a detailed study of the targeted gene expression and function; however, some clues might be provided by the nature of the malformations themselves if they are consistent with a single developmental origin (e.g. neural crest derivatives) or reminiscent of an established congenital syndrome.
It should be possible to resolve these possibilities in the future using mass spectrometry under conditions that provide quantitative information on modification occupancy.
Additional genetic analyses, including allelism tests and gene cloning are needed to resolve these possibilities.
Functional studies of the mitochondrial sdh4 gene, or demonstration of functional transfer of sdh4 to the nuclear genome, will help resolve these possibilities.
Further experiments are needed to resolve these possibilities.
Further experiments are required to resolve these possibilities.
With the current data set, we cannot resolve these possibilities.
Sequencing of LNX homologs from additional basal metazoan lineages may resolve these possibilities.
Continued collection of patient samples and further investigation of results would be necessary to resolve these possibilities.
Further data will be needed to resolve these possibilities.> -wrap-foot> Viabilito to adulthood of each genotype combination at 25°C.
To resolve these possibilities we needed to examine the levels of ERα protein expression in the different clones and we needed to carry out more effective ERα knockdown.
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