Sentence examples for repression cell from inspiring English sources

We discuss the potential applications of the engineered repressor strategy that relate to target gene analysis, mechanisms of repression, cell regulation, and possible anti-viral and cancer therapy.

Together, this suggests that the information driving the quantitative effects of Hox proteins on Wnt repression, cell ingression, and elongation is built in the structure of the proteins themselves rather than reflecting the actual amounts of Hox proteins present.

For mTOR repression, cells were incubated with pp242 (1 μM) or rapamycin (1 μM or 200 nM for GH3 cells) for different time periods (6 h for Fao, 12 h for MEFs, 24 h for 3T3-L1 and GH3).

Paradoxical results on Tcf-4-regulated OPN expression in MCF10AT (activation) and Rama37 (repression) cells were shown to be a result of differential Wnt signalling competency in MCF10AT and Rama37 cells.

For setting up the repression, cells were peletted down in mid-log phase and shifted to pre- warmed 0.15X YEP medium without any carbon source and incubated for 4 hours [ 22].

As shown in Figure 8, depending on the levels of c-MYC repression, cells with ~50% c-MYC knock-down (sh1) displayed significant reversal in target gene expression such as LAMC2 and VAV3, but cells with ~25% c-MYC knock-down (sh2) only showed relatively minor change.

This indicates a strong repression of cell wall synthesis, cell wall modification, pectin degradation, cell expansion and cell wall turnover.

Immunofluorescence showed that an increase in apoptotic cells and repression of cell proliferation occurred in the combination treatment with fulvestrant and paclitaxel, compared with paclitaxel alone.

In lymphoid cells, this results in repression of cell cycle progression through cyclin-D3 and C-MYC [ 7], and cell death through the activation of apoptosis.

Through the repression of cell wall modifying enzymes, cell growth is directly constrained to inhibit germination.

This rule states that high power intensities cause a repression of cell activity or even cell damage at higher dosage.