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GeneDistiller includes the following data: Genes, gene positions, gene RIFs, gene ontology, cellular localisation of gene products, transcripts, exons, OMIM reports, mouse phenotypes, protein-protein interactions, gene expression data, protein domains, SNP markers, STR markers.
Thus, the study, for the first time, reports mouse iPS cell lines generated on human feeder cells, providing a reliable tool for further dissecting the molecular mechanisms of nuclear reprogramming.
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Consistently with the previous reports, mice with two weeks of rapamycin treatment had characteristics of metabolic syndrome.
Consistent with previous reports, mice transplanted with p210 BCR/ABL1 exhibited B-cell lymphocytosis when killed at day 20 and 38 post BMT (Table 2, p210 BCR/ABL1 mice 1 6).
To evaluate the virulence of FAM20LU, we used a previously reported mouse model [18] and challenged CD46 transgenic mice i.p. with 108 FAM20LU.
As already reported, mouse and human NSCs can be reprogrammed with Oct4 and Klf4 only, since NSCs intrinsically express Sox2 and c-Myc [7], [8].
If so, can memory function be enhanced via transgenic overexpression of NR2B in another species other than the previously reported mouse species?
There are few previously reported mouse models of lung SQCAs.
All reported mouse studies were approved by the Institutional Animal Care and Use Committee at the Saban Research Institute of Children's Hospital Los Angeles.
To compare ESC miRNA profiles between species, we retrieved previously reported mouse and human ESC miRNA profiles.
Together, these reported mouse models might have a different TDP-43 pathology from that found in patients with ALS.
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