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Moreover, the presence of melancholia has been reported to predict a poor response to psychotherapy and placebo and a relatively good response to antidepressants and electroconvulsive therapy [ 9].
The presence of two or more AKI risk factors has been previously reported to predict a cohort of patients at high risk of developing AKI [ 24].
Presence of WMH has been reported to predict a three-fold increased risk of stroke and two-fold increased risk of mortality in the general population [ 4].
Higher TFF3 mRNA levels have previously been reported to predict a worse survival outcome of patients with ER+ MC treated with tamoxifen [ 14].
Tissue IGFBP-3 concentrations have been reported to predict a reduced OS, but this was not associated with breast cancer recurrence [ 25].
9– 16 Older age is reported to predict a poor prognosis, 9– 11 and several previous reports have shown a relationship between male sex and poor prognosis, 12 although the data have been inconsistent, especially for pediatric DCM.
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For example, failure of closed-chest resuscitation to increase the PETCO2 above 10 mmHg has been reported to predict an extremely low likelihood of restoring spontaneous circulation [ 6, 7].
Overt diabetes (1, 10) as well as diabetes defined based on 1-h postload glucose (11), FPG levels (12, 13), or nonfasting glucose levels (7, 14) have been reported to predict an increased risk of stroke.
The same study found that the gene expression signature that results from ectopic overexpression of NDN is also reported to predict prognosis in a human breast cancer cohort (Crawford et al, 2008).
While the absolute baseline serum mesothelin level has not been reported to predict for treatment response a number of trials have demonstrated that a fall in the mesothelin level with treatment correlates well with radiological response rate and overall survival [ 24, 25].
Both markers of subclinical inflammation were reported to predict cardiovascular events in a range of studies (1– 3).
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