MAP1D is over-expressed in colon cancer [ 12], but no study has reported the expression of PDF in cancerous compared to normal tissues.
In addition, artemin was shown to be located in the smooth muscle cells of dural vasculature, which was in agreement with a study by McIlvried, who firstly reported the expression of artemin in the dura mater of rats [11].
In a follow up report, (Rasala et al. [2011]) reported the expression of 14FN3, a potential antibody mimic, using a line that lacked the psbA gene, and hence, was non-photosynthetic.
Domitrović et al. recently reported the expression of 4-hydroxy-2-nonenal (4-HNE), one of the lipid peroxidation products, in the kidney of cisplatin-treated mice by immunohistochemical study [24].
In Figure S2B is reported the expression of MYC mRNA after silencing using siMyc Mix.
Previous studies have reported the expression of UPs and their assembly into AUM is essential for the maintenance of urothelial barrier function [15] [18].
We have previously reported the expression of endothelial NOS (eNOS) and inducible NOS (iNOS), but not neuronal NOS, in rat mesangial cells [29].
Frolich et al. recently reported the expression of Nrf2 and GST mu family genes were significantly decreased in TRAMP prostate tumor, and linked this phenomenon to increased oxidative stress and DNA damage in prostate cancer.
Several evidences indicate that sex hormones affect platelet physiology, and it has been reported the expression of estrogen receptor (ER) beta and androgen receptor (AR) in human platelets [37].
Previously we have reported the expression of P450scc and its electron transfer partners in the skin and demonstrated the capability of skin mitochondria and intact melanoma cells to transform cholesterol to pregnenolone [10].
While a number of studies have reported the expression of activity-dependent reductions in strength at inhibitory synapses in area CA1 [11] [14], plasticity specifically at the feedforward inhibitory synapses has never been demonstrated.
