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All studies stated the time horizon for costs and benefits and most (35 or 90%) reported a time horizon of one year or less.
Interestingly, a study exposing PNECs to CSE for 1 h, 2 h, and 4 h reported a time and dose-dependent cytotoxicity of CSE [ 33].
Among women who reported never use at baseline and ever use at follow-up, approximately 17.4percentt reported a time for their most recent mammogram (at follow-up) that was inconsistent with never use at baseline.
A 76-patient phase II study reported a time to progression of 8.0 months, a median survival of 14 months, and a response rate of 25% using the same regimen [ 23 ].
Based on data from the Beaver Dam Health Outcome Study, Malter et al. [ 14] reported a time tradeoff utility level of 0.89 for patients with satisfactory outcomes and 0.56 for patients with unsatisfactory outcomes after treatment of lumbar disc herniation.
Predominant breastfeeding for four months refers to those mothers who chose response one for question two (only breast milk) and reported a time period of more than 120 days.
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We recently reported a time-sensitive, cooperative, anti-tumor effect elicited by radiation (RT) and intra-tumoral-immunocytokine injection in vivo.
We previously reported a time-course analysis of a 1-day span using a rice 22 K microarray, in which we discovered OsIRO2 (Ogo et al. 2006).
A limitation is our single post-exercise assessment of cTnI, as previous observations reported a time-dependent change in cTnI after a marathon (Middleton et al. 2008).
Several previous studies reported a time-dependent loss of CA signals on cytoplasmic structures positive for the HIV-1 accessory protein Vpr in HeLa- or HOS-derived cell lines infected with VSV-G pseudotyped VLPs (McDonald et al., 2002; Hulme et al., 2011), suggesting gradual capsid uncoating during reverse transcription.
We recently reported a time-dependent and concentration-dependent desensitization of DA D2 receptors requiring concurrent activation of D1-like and D2-like receptors which we called DA inhibition reversal (DIR; Nimitvilai and Brodie, 2010), and this desensitization is mediated by phospholipase C and the protein kinase C (PKC) pathway (Nimitvilai et al., 2012).
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