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To determine if IKKγ isoform expression affects RSV replication, the expression of RSV proteins were detected in Western immunoblot using a pan-anti-RSV Ab.
To elucidate antiviral effect of amiR-53R-1 on RGV replication, the expression of 53R gene was monitored in GCO cells transfected with pSM155-amiR-53R-1 pSM155-amiR-53R-1 pSM155-amiR-53R-1 pSM155-amiR-53R-1andfectinfected
Although the data suggested that FAS II is necessary for intra-erythrocytic replication, the expression of FAS II enzymes has not been studied throughout the complex infection cycle of the parasite and their importance in parasite progression throughout the life cycle remains unknown.
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To investigate the mechanism by which HAT1 or KAT8 affected HBV replication, the expressions of HBV replication-related transcription factors Foxa1, Foxa2, Foxa3, CEBP, HNF1A, HNF1B, HNF4A, PPAR, FXR, FTF, and PPARGC were detected using qRT-PCR analysis.
Although this study contains a replication of the genetic findings, it remains limited due to the lack of replication of the expression findings, thus warranting further studies.
This vector is based on the pY37 previously described, harbouring a S11 Kluyveromyces origin of replication, and the expression of GAP1 is under the control of GAL1.
In addition to cell cycle control, there appears to be a correlation between initiation of chromosome replication and the expression of the nrdAB genes (reviewed in [18]).
Thus, in this study we have demonstrated that a cellular transcription factor that is induced by IFNβ and IL-10 is important in the brain for regulating virus replication and the expression of pro-inflammatory cytokines that are strongly linked to CNS inflammation and HIV-associated CNS disease.
In the case of double-stranded DNA viruses, genome replication represses the expression of early proteins.
MtSSB is necessary for mtDNA replication and the expression of mtSSB is strictly regulated and correlates directly with mtDNA content (Schultz et al, 1998).
Cigarette smoke extract promoted EBV replication, induced the expression of the immediate-early transcriptional activators Zta and Rta, and increased transcriptional expression levels of BFRF3 and gp350 in the lytic phase [ 3].
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