These studies begin to unravel requirements for robust virus replication and the relationship between increased virion production and host cell viability.
This study will characterize HIV-1 subtype C viral load set point in heterosexual infection in South Africa, the role of specific T cell immune responses in the control of replication, and the relationship between viral set point, CD4+ T cell count trajectory and disease progression.
After careful and extensive replication studies, the relationship between candidate epigenetic alterations and schizophrenia pathology must be validated using cellular and animal models.
A controlled experiment and three replications examined the relationship between a person's age as a status characteristic and the value placed on that person as a potential group member.
Due to the many comparisons, cautious interpretation and replication of these relationships are warranted.
As observed previously for virophages and Polintons [ 29], analysis of the PLV genes implicated in genome replication revealed complicated relationships suggestive of complex evolution.
In the present study, we sought to obtain a greater understanding of the relationship between replication timing programs and human chromosomes by analysis of the timing of replication of a single human chromosome 11 that had been transferred into the Chinese hamster ovary (CHO) cell line by chromosome engineering.
Next, we investigated the relationship of replication initiation to gene expression level.
The aim of this study was to describe the mechanisms of reversion of RT mutations to wild-type viral population during STI and the relationship with replication capacity (RC) as a measure of fitness.
To examine the relationship of replication initiation with transcription, we initially compared the location of known transcription start sites (TSS) contained in our array to the pattern of origin peaks obtained in MCF-7.
We traced the relationship between replication timing and the evolutionary age of duplicated genes.
