Additionally, advances in viral based therapy have led to the use of condition replicative adenovirus controlling the replication of the virus mainly through transcription regulation by using tumor specific radioinducible promoters [23, 24].
For both inoculation routes the highest viral loads were observed in lymphoid organs, indicating a progressive infection and replication of the virus in these organs, whereas calpox virus replicated less efficiently in organs of the digestion tract and the brain.
A prevailing opinion is that replication of the virus must be suppressed as much as possible.
Cocaine greatly enhanced replication of the virus and increased the number of human cells it infected and killed.
Anti-H.I.V. drugs suppress replication of the virus, which should give the functioning parts of the immune system a chance to eliminate remaining virus.
Inhibitors can prevent HIV from entering cells or block steps in the replication of the virus.
Prostratin is thought to activate a protein that promotes replication of the virus.
After the blender had separated the DNA and the protein, an examination showed that only the DNA had entered the bacteria at the time of infection; this DNA then led to the replication of the virus.
Many RNA viruses such as HIV use a specialized enzyme called reverse transcriptase that permits replication of the virus through a DNA intermediate.
In some cases, as in herpes infections, the viral nucleic acid may remain in the host cell without causing replication of the virus and damage to the host (viral latency).
To stop the replication of the virus, we developed highly potent tetrapeptidic HTLV-I protease inhibitors.
