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Data may be distributed by fragmentation and replication of tables.
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We observe greater variation in the number of detected N signals for the cell lines compared to the reference, indicating that biological variation is larger than variation resulting from technical replication of measurements (Table 1).
In our Slovak RA case-control sample (Table 1), we sought direct replication of these SNPs (Table 2).
HDACs are involved in the regulation of the replication of numerous viruses (Table 2).
This is included in Anna's response (4.Shrimp.C), but Beth does not consider replication of treatment (4.Shrimp.D.ii; Table 2, area of difficulty 4h).
Although the number of already known (3 loci) and replicated loci (1) was not large enough to build a predictive model, we inspected the features of both groups of AREDS SNPs (replicated and non-replicated) for insights that might facilitate efficient selection of SNPs for replication (Table S1 and Table 5).
Two additional cohorts contributed to replication of the results (ESM Tables 3– 5).
As shown in Figure 2 B, a higher proportion of VET children presented negative or indeterminate results: seven of the 14 children in the VET group (50%) vs only one of the 9 ET children (11.1 %), probably as a result of early and sustained control of viral replication (Table 2 and Supplementary Table 1).
In analyzing the 147 evolutionary literature breakpoints, the enrichment and distance analyses produced highly significant correlations for tRNAs and LTRs and for origins of replication (supplementary table S10, Supplementary Material online).
The 1625 BPSs that accumulated in the MutL− strain were collected into 46 bins, each bin approximately 100 kb in size, starting at the origin of replication (see Table S1).
Many enzymes potentially involved in the replication of viral DNA are predicted (Table 3, and EU304328).
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