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This study aimed to develop ultra-fine blast furnace slag based geopolymer as concrete repairing agent.
Most of the treatment groups except methanol extract (200 mg/kg bw) served as repairing agent helped in normalizing the serum CK-MB level in hepatic damage while ethyl acetate 200 mg/kg bw and chloroform 100 mg/kg bw played the most significant role in restoring the serum CK-MB (Table 3).
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In the present study we have designed novel ceramide analogue 14S24 ((S -2-tetracosanoylamino-3-hydroxypropionic acid tetradecyl eS -2-tetracosanoylamino-3-hydroxypropionicnt.
Dissipative particle dynamics (DPD) simulations were applied to investigate the coating repair agent dicyclopentadience (DCPD) in pH-sensitive micelles.
The crack was more and more difficult to be repaired with the increase of average crack width and the repair ability of microbial repair agent was limited for specimens with crack width up to 0.8 mm.
The self-repair microcapsules in the epoxy resin would break as a result of microcrack expansion in the matrix, and letting out the strong repair agent to recover the mechanical strength with a relative healing efficiency of up to 140% which is a ratio of healed property value to initial property value or healing efficiency up to 119% if using the healed strength with the damaged strength.
These results demonstrate that this new bioactive material has many properties that would allow it to be effectively applied directly to a wound to accelerate reepithelialization of non-healing, chronic wounds and has significant potential as a wound repair agent.
Experiments where the repair agent was co-transfected with the target gene were useful because the repair frequency was generally 10 to 100-fold higher than gene targeting at the chromosomal level [ 50].
Episomal gene correction assays were performed as follows: plasmid DNA- and the gene repair agent (ssODN and dsDNA)- complexes were generated separately in Optimem-I medium with Lipofectamine Reagent using a cationic lipid/DNA (w/w) ratio of 4 1.
The trajectory and time course of RNFL axonal loss seen with OCT following an episode of acute optic neuritis is important for determining the 'window of opportunity' within which a neuroprotective or repair agent might be administered in a clinical trial setting.
When rAAV vectors were used as repair agent, the culture medium was removed one hour after the transfection step (which allowed for the delivery of the mutated reporter construct) and replaced by the infection mixture consisting of diluted rAAV-2/1 vectors (0.25 ml medium/well).
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