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H2O often results in irreversible renal damage.
Second, cisplatin induces renal interstitial inflammation and chronic interstitial fibrosis, leading to irreversible renal damage.
Nonpulsatile perfusion during cardiopulmonary bypass can induce renal damage.
Renal damage is more frequent with new-generation lithotripters.
Most individuals who survive completely recover from acute renal failure, but residual renal damage persists in some persons.
Haptoglobin is an acute phase protein that binds haemoglobin, thus preventing iron loss and renal damage (Wassell, 2000).
Clearly, primary vascular disease disease affecting the blood vessels could equally well be a cause of renal damage.
In turn, the infiltrating leukocytes contribute to renal damage by releasing inflammatory and profibrotic factors.
Myoglobin has been identified as the primary muscle constituent contributing to renal damage in rhabdomyolysis.
Cardiopulmonary bypass (CPB) represents a specific risk factor for renal damage during coronary revascularization.
Patients with severe VUR showed a higher probability of renal damage than those with nonsevere VUR.
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