Sentence examples for remarkably demonstrated from inspiring English sources

The interest of muscle biopsy has been remarkably demonstrated by Bradley et al. [54], by Puthucheary et al. [46], and by Hermans et al. [43], who thereby assessed the role of mitochondrial dysfunction, proteolysis, and autophagy, respectively.

The cellular arm of the host immune system has been associated with partial virological control, remarkably demonstrated in studies of CD8+ T-cell depletion; CD8 T-cell immune escape and by the association between specific HLA class I alleles and favourable HIV disease outcome [21], [24].

The interest of muscle biopsy has been remarkably demonstrated by Bradley et al. [ 54], by Puthucheary et al. [ 46], and by Hermans et al. [ 43], who thereby assessed the role of mitochondrial dysfunction, proteolysis, and autophagy, respectively.

In summary, our study remarkably demonstrates that transduction of pancreatic exocrine cells requires lentiviral vectors in vitro but adenoviral vectors in vivo.

The dispersion of MMT and the interfacial interaction between PDBIIVP and MMT were improved remarkably, as demonstrated by X-ray diffraction, transmission electron microscopy, rubber process analyzer (RPA) results and swelling tests.

Remarkably, S9 demonstrated similar inhibition in drug resistant cancer cells and respective parental cells, with the resistant factors around 1(Fig 6B).

Remarkably, we demonstrated that Prdm13 in the DMH is important to maintain sleep quality and body weight associated with adiposity.

Remarkably, we demonstrated that miR-16 is significantly downregulated by MPA treatment in an in vivo setting.

Remarkably, we demonstrated that administration of exogenous TGF-β produced an increment on the level of intracellular TGF-β expressed by tumor cells, inhibiting proliferation and augmenting apoptosis.

Remarkably, they demonstrated that this resistance was due to genetic variation in one particular form of IRG protein, named Irgb2-b1: they found that certain alleles of Irgb2-b1 appeared to inhibit the activity of the effector molecules secreted by the parasite, which themselves have evolved to inactivate the IRG system.

Remarkably, we demonstrated that the siRNA-mediated knock down of GSK3α and of both the GSK-3 isoforms but not of GSK-3β alone, was associated with the same changes of AKT and MCL-1 levels as seen with the inhibitors.