However, on average, 30 to 50% of the predicted proteome remains uncharacterized while for the remaining set only general predictions are made.
However, the role of BRD9 in BAF complex targeting and activity remains uncharacterized.
However, its role in the regulation of hepatic TG homeostasis remains uncharacterized.
Overall immune responses to O. ostertagi are poorly understood, and the early innate immune response to this parasite remains uncharacterized.
Previous studies have tried to show the catalytic residues, nevertheless, the accurate architecture and molecular behavior of its emitter site remains uncharacterized.
Antidepressant usage in prodromal Huntington Disease (HD) remains uncharacterized, despite its relevance in designing experiments, studying outcomes of HD, and evaluating the efficacy of therapeutic interventions.
The genetic compensation response (GCR) has recently been proposed as a possible explanation for the phenotypic discrepancies between gene-knockout and gene-knockdown1,2; however, the underlying molecular mechanism of the GCR remains uncharacterized.
Similarly, various fluorescently labeled lipid analogs have been used to study membrane organization and function in vitro, although the biological activity of these probes in relation to their native counterparts often remains uncharacterized.
The SFT11/12 double mutant remains uncharacterized.
The mechanism leading to changes in the superstructure of endothelial cells exposed to ischemia and reperfusion remains uncharacterized.
Although a limited sweep is proposed to alleviate this problem, the tradeoff between accuracy and complexity remains uncharacterized.
