Although some of the molecular features related to these cell behaviors have been described, the underlying mechanisms, at a global level, remain undefined at midgestation.
However, many cases of COX deficiency remain undefined at the molecular level.
The common mechanisms between these syndromes and STAT3 and DOCK8 deficiency remain undefined at present.
However, the protein protein interaction surfaces, catalytic mechanisms, and overall architecture of these macromolecular machines remained undefined for quite some time.
While the molecular targets of MEKi leading to increased MHC-I expression remain undefined, this suggests that it may interact with signaling downstream of the IFNγ receptor.
However, the mechanisms of cadmium carcinogenesis at any site remain undefined, and additional models at all levels of biological complexity, including models of pancreatic cancer, are clearly needed.
However, the details of this relationship remain undefined.
However, the mechanisms by which progesterone withdrawal manifests this effect remain undefined.
The specific etiology and mechanisms of the respiratory disease in this building remain undefined.
The mechanisms involved in this repair process remain undefined.
The nature of the innate mechanisms that govern this portion of the response remain undefined.
