Sentence examples for release was prevented from inspiring English sources

Massive radiation release was prevented.

That the effect of kisspeptin on LH release was prevented by pretreatment with GnRH antagonist further points to the action of kisspeptin upstream of GnRH (Gottsch et al., 2004; Shahab et al., 2005).

Interestingly, LDH release was prevented significantly in apoptotic cells without AMN by colchicine treatment when caspases were inhibited by z-VAD suggesting that caspase-mediated cleavage of plasma membrane and cellular cortex proteins was essential for releasing cell content.

To allow accurate quantitation of cells with cytoplasmic cytochrome c, the apoptotic consequences of cytochrome c release were prevented with the caspase inhibitor z-VAD-fmk.

We have previously shown that both cAMP superactivation and the cAMP-dependent increase in GABA release are prevented in RMOR mice that have been treated chronically with morphine (He et al, 2009; Madhavan et al, 2010).

The ethylcellulose coating, when applied organically, can accommodate the swelling matrix to a greater extent compared to aqueous coating and hence, coating rupture and subsequent drug release is prevented.

Also, infusion of GLP-1 to pharmacological levels fails to stimulate insulin-mediated glucose disposal in healthy volunteers or under experimental conditions where its effects on endogenous insulin release are prevented (as in type 1 diabetic patients) (rev. in 79).

In contrast, in the presence of a fast Ca2+ chelator (10 mM BAPTA), this local Ca2+ cycling is prevented (no CICR triggered), such that ICa inactivation is slower (50) and reflects a small rise in local Ca2+ concentration close to the mouths of channels due to Ca2+ entering via the LTCC only (SR Ca2+ release is prevented) (53).

Accordingly, the mutation in the 171/172 amino acids results in a mutant with remaining high Ca2+ sensitivity for achieving uptake of entering Ca2+, while the contribution of this UCP3 mutant to mitochondrial uptake under high Ca2+ conditions (i.e. intracellular Ca2+ release) is prevented.

As vitamin D3 acts as an inhibitor of IL1-β release, its activity was prevented by exogenous IL1β and was dependent on the expression of VDR on macrophages (Fig. 4A, Fig. 4B) and not on tumor cells (data not shown).

When the release of synaptic transmitter was prevented by a high-magnesium, low-calcium Ringer solution, stimulation of preganglionic nerve fibers did not increase cyclic GMP in the ganglion.