Sentence examples for release impairment from inspiring English sources

CDKN2A/2B encodes for p16INK4a, a tumor suppressor influencing pancreatic β-cell proliferation [37], [38].It is possible for a causal variant situated in CDKN2A/2B to increase the susceptibility of T2DM through β-cell mass reduction and subsequent insulin release impairment in the sates with increased insulin demand.

These preliminary results suggest the possibility of a new, "softer" pharmacological approach to ADHD: hydrophobic mode matched peptide allosteric augmentation of the activity of indigenous dopamine with respect to D2DAR mediated function, in place of stimulant drug-induced presynaptic dopamine release or impairment of dopamine uptake.

Departments are responsible for ensuring that all such equipment is appropriately tagged and that any changes are reported to the Controller's Office including the sale, transfer, release, disposal or impairment of any equipment under their jurisdiction.

Departments are responsible for the safeguarding of all equipment assigned to their school, unit, and or department; for ensuring that all such equipment is appropriately tagged and that any changes are reported to the Controller's Office including the sale, transfer, release, disposal or impairment of any equipment under their jurisdiction.

Cell transplantation started with clinical trials in patients with PD, in which transplantation of human fetal mesencephalic tissue rich in dopaminergic neurons was found to normalize dopamine release and reverse impairment in cortical activation [7, 8].

The anemia of CKD is commonly attributed to lack of renal erythropoietin release and subsequent impairment of erythropoiesis [ 266, 267].

Severe complications of end stage renal disease include anemia [ 1, 2], which is at least in part the result of restricted renal erythropoietin release and subsequent impairment of erythropoiesis [ 3, 4].

Hearing loss or impairment.

Moreover, the impairment in release that we observed for the K26A mutant, and the increase in spontaneous release caused in rescue experiments on complexin knockdown neurons by a K26E, L41K, E47K mutation (charge change = +1) (Yang et al., 2010) also correlate in general terms with this model.

This suppression was associated with a prominent increase in glucose-stimulated insulin release and thus the impairment of glucose-induced insulin response in GK islets was efficiently counteracted (Fig. 4a).

Nevertheless, we cannot exclude an impairment of release.