Asthma morbidity has been associated with exposure to several ambient air pollutants, e.g., sulfur dioxide (SO2), particulate matter (PM2.5) and nitrogen dioxide (NO2) [ 3- 7], and recently its relationship to traffic-related pollutants has received considerable attention.
Not being in the right place at the right time causes lots of problems and inconveniences — from long-distance relationships to traffic-filled commutes, rural isolation to scarce access to art and entertainment.
The Panel concluded that the evidence was "sufficient" to infer a causal relationship between exposure to traffic and exacerbation of asthma and "suggestive but not sufficient" to infer a causal relationship with onset of childhood asthma, non-asthma respiratory symptoms, impaired lung function, and total and cardiovascular mortality.
Another focus of research on cell coverage is the inverse relationship of coverage to traffic load in a cell.
We estimated the relationship between exposure to traffic and occurrence of AMI using logistic regression, and we adjusted for the following potential confounders: age, sex, section of the study area, point sources emissions of particulate matter with aerodynamic diameter < 2.5 μm, area socioeconomic characteristics, and percentage of open space.
Also accounted for are the exposure to lead paint, proximity to traffic, and geographic relationship to hazardous sites.
Few published studies have analyzed the effects of traffic in relationship to combinations of respiratory outcome such as asthma symptoms and persistent cough or phlegm.
To define KChIP1 vesicles and the traffic pathway followed by Kv4/KChIP1 traffic, we examined their relationship to potential SNARE (soluble N-ethylmaleimide-sensitive fusion protein-attachment protein receptor) proteins mediating the trafficking step.
This study investigated the relationship between living close to traffic and ever dispense of prescribed oral antihistamines or nasal anti-allergic medication, among young children.
Better phenotypic characterization of atopy both in the study children and in their parents and of allergen exposure in children would be useful to interpret the relationship of these characteristics to traffic and asthma.
