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As a consequence, dopamine release from serotonin nerve terminals will be poorly regulated, resulting in uncontrolled, excessive swings in dopamine release.
Under drought, both structural growth (sink) and assimilation (source) processes are down regulated, resulting in changed source-sink relations that may depend on environment and genotype.
As a result of the lack of these mechanisms, dopamine release from serotonin nerve terminals in PD may be poorly regulated, resulting in uncontrolled excessive swings in dopamine release (called release of 'false transmitter').
In normal tissues, Hh-induced progenitor-cell proliferation is transient and tightly regulated, resulting in acute epithelial repair and regeneration after injury (Beachy et al, 2004).
The multicopy P1 allele is epigenetically regulated, resulting in the phenotype P1-wr (white pericarp/red cob) due to hypermethylation [ 34, 35].
Within this template, chondrocyte proliferation, differentiation (hypertrophy) and apoptosis are precisely regulated, resulting in endochondral bone growth and ultimately replacement of cartilage by bone tissue.
During cell differentiation, groups of genes driving specific developmental processes are often commonly regulated, resulting in similar expression patterns in time course-data.
In addition, the multicopy P1 allele is epigenetically regulated, resulting in the phenotype P1-wr (white pericarp/red cob) due to hypermethylation [ 34, 35].
It appears that binding of the p52 Bcl3 complex to the G/C- and A/T-centric κB sites is differentially regulated, resulting in kinetic instability of complexes involving G/C-centric sites but not those involving A/T-centric sites.
Stomatal abundance in different plant surfaces and environments is regulated, resulting in variable stomatal numbers and distribution patterns in mature organs (Bergmann and Sack, 2007; Casson and Hetherington, 2010, and references therein).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com