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Past studies have explained that the platelet aggregation and adhesion is regulated by release of vWF (Vischer 2006), and subsequently, the high level of vWF has been attributed indirectly to atherosclerosis and thrombogenesis (Lip and Blann 1997; Vischer 2006).
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Release of follicle-stimulating hormone and luteinizing hormone by the pituitary is regulated by the release of gonadotropin-releasing hormone (GnRH), the hypothalamic hormone.
LH hormone, produced in the pituitary, is regulated by gonadotropin-releasing hormone (GnRH) released from the hypothalamus [ 35].
The HPA axis is regulated by corticotrophin-releasing hormone (Crh) released from neurosecretory cells of the hypothalamic paraventricular nucleus.
It would then be regulated by a release of insulin from the hydrogel system.
In the body under physiological conditions, many vital functions are regulated by transient release of bioactive substances at a specific time and site.
Cell communication represents a dynamic mechanism regulated by the release of factors able to influence cell fate, function and plasticity.
This cellular response is regulated by SLPI release.
The refractory period is regulated by sperm release.
EPCR can be regulated by proteolytic release from the cell surface to form sEPCR [ 12, 13].
These results disclose that the radiation-induced NO is regulated by the release of mitochondrial cyt- c.
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