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Significant questions still remain, however, as to the genetic and epigenetic signals that regulate the fate of stem cells.
Thus the position of the planets and their interrelation with the signs of the zodiac, regulate the fate of both individuals and nations.
It would be interesting to examine the cell properties after exposure to the HMF to investigate whether this magnetic field could be applied to regulate the fate of NSCs.
What processes regulate the fate of newborn DGCs, how do they integrate into the existing neuronal circuitry of the hippocampus, and what are the physiological/behavioral roles of DG plasticity?
The objective of the present study was to investigate how pore size and surface chemistry of three-dimensional (3D) porous scaffolds regulate the fate of mesenchymal stem cells (MSCs) in vitro in combination.
Numerous mRNA-binding and regulatory proteins within mRNPs finely regulate the fate of bound-mRNAs.
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In addition, recent studies have proven that balanced mitochondrial dynamics and morphology are crucial to the maintenance of tissue homeostasis: mitochondrial dynamics regulates the fate of stem cells.
These findings indicate that AMH differentially regulates the fate of Sertoli cells in vitro by promoting proliferation at low concentrations and apoptosis at high concentrations.
Attachment of ubiquitin (Ub) or ubiquitin-like (Ubl) modifiers is a reversible post-translational modification that regulates the fate and function of proteins.
Extensive studies on NSCs revealed the mechanisms of asymmetric division which involves a number of polarity and fate determinants whose spatiotemporal distribution regulates the fate of two daughter cells.
Here we focus on the emerging roles of galectins, a family of highly conserved glycan-binding proteins in regulating the fate and function of regulatory immune cell populations, both of lymphoid and myeloid origins.
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