Sentence examples for regulate inflammatory response from inspiring English sources
"regulate inflammatory response" is a correct and usable phrase in written English
It is typically used in scientific or medical contexts to describe the process of controlling or managing the body's reaction to inflammation. Example: "The new medication was found to effectively regulate inflammatory response in patients with rheumatoid arthritis, reducing their symptoms and improving their quality of life."
Monocytes or macrophages, for instance, upregulate anti-inflammatory SH2-containing inositol phosphatase-1 (SHIP1) and regulate inflammatory response.
Regulatory T cells (Tregs) are known to regulate inflammatory response and suppress alloreactive T cells; the exact mechanism is unknown so far.
This way, innate immune signalling pathways are triggered and the secretion of several factors that regulate inflammatory response allows the expression of proinflammatory cytokines and chemokines [ 52, 53].
For example, it may regulate inflammatory response such as chemokine release (for example interleukin (IL -6, IL -6 to promote/inhibit tumor growto in a promote/inhibittumorashion [ 27].
These genes included those for chemokines such as CXCL-14 [chemokine (C-X-C motif) ligand 14] and CCL-5 [chemokine (C-C motif) ligand 5], suggesting that some genes that regulate inflammatory response to LA tend to be inhibited in SH and SHHF rats but not in WKY rats.
Given the ability of PBA and TUDCA to regulate inflammatory response, cell death and cell proliferation in various diseases, we examined the effects of these ER stress inhibitors (PBA and TUDCA) on hepatic injury and liver regeneration in steatotic and non-steatotic livers in PH+I/R.
EP300 and PCAF also regulate inflammatory responses through their regulation of cyclooxygenase-2 (COX2) expression.
Endogenous release of adenosine triphosphate (ATP) in the liver activates purinergic P2 receptors (P2R), which regulate inflammatory responses, but little is known about the roles of these processes in the development of acute hepatitis.
This may have important implications in vivo since TGF-β and IL-10 regulate inflammatory responses.
These cells, originally identified in tumor bearing hosts, have been shown to possess potent suppressive functions and regulate inflammatory responses [24], [25].
The healthy gut is able to regulate inflammatory responses to commensal bacteria and food antigens whilst maintaining the capacity to respond to pathogens [1], [2], [3].
