Sentence examples for regulate disease severity from inspiring English sources

Several studies have reported that TLR4 and TLR9 may regulate disease severity of EAE, but the detailed functions of TLRs during MS/EAE are still controversial (Kerfoot et al, 2004; Marta et al, 2008; Prinz et al, 2006).

In the IBD field, key areas of research include improving our understanding of: how the host genome regulates disease susceptibility; how interactions between the host microbiome and host immune system contribute to disease induction; and how cellular and molecular events at the epithelial barrier interact with the host immune system to regulate disease severity and progression.

Cia5a is a locus on rat chromosome 10 that regulates disease severity and joint damage in two models of rheumatoid arthritis, collagen- and pristane-induced arthritis (PIA).

Numerous cytokines, both proinflammatory and anti-inflammatory, have been detected in the ST of RA, and the balance between these opposing cytokine activities regulates disease severity [ 10].

We speculate that the genotype of the relevant S. Typhi organisms may also play an important role in regulating disease severity.

Dengue serotype is known to influence disease severity in Singapore35.

This conclusively shows that IL-6 regulates chronic disease severity by regulating the early immune response following viral infection.

We examined how genome-wide microRNA expression is altered with regional emphysema severity and how these microRNAs regulate disease-associated gene expression networks.

To determine if IL-6 production during the early immune response regulates the chronic disease severity, recombinant IL-6 (rIL-6) or DMEM was administered to IL-6KO mice at days 0, 1, 2 and 3 PI and the chronic disease severity was observed at 28 days PI.

This demonstrates how alterations in TCS and QS regulate the balance between disease severity and persistence, i.e. acute (dispersed) or chronic (persistent, biofilm-forming) infections.

Disease development and proliferation of GMI1000 in Arabidopsis was not SA-dependent, but inactivation of ET-related signaling pathways resulted in decreased symptom development in susceptible plants, indicating that ET-regulated defenses reduce disease severity [29].