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Sir, We read with great interest the letter by Coon et al. [1], recently published in this journal and we would like to focus our attention on the following concerns regarding this report: 1. Burning mouth syndrome is a chronic, idiopathic oral mucosal pain/discomfort, in which no clinical lesions or systemic diseases or other causes, especially drugs are identified [2, 3].
Regarding this report, I was extremely pleased that the rapporteur embraced the views of people from the various groups and that we reached unanimity.
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Details regarding this study will be reported separately.
A consent form regarding this case report has been signed by the patient, and has been submitted separately.
Regarding this possibility, previous reports have demonstrated that the two cell-types are unlikely to be associated by direct neuronal connection, but instead by gap junctions [24] [27].
Regarding this, various studies reported changes in the mechanical properties of decellularized tissues.
The specificity and sensitivity are reportedly high, but there is some disagreement regarding this between different reports [ 3- 6].
Regarding this phenomenon, some reports have been indicated that the antimicrobial activity may be due to the produced acid [ 26, 27].
Regarding this point, previous reports suggest postsynaptic localization of the 5-HT2A Rakictors (Rakic et al. 1988; Lópéz-Giménez et al. 2001; Miner et al. 2003).
report regarding this sector".
Subsequent to the CAO report regarding this project, CGPL voluntarily agreed on an action plan that includes a framework of studies and measures.
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CEO of Professional Science Editing for Scientists @ prosciediting.com