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The EMD groups showed more reduction in probing pocket depth with concomitant gain in periodontal attachment level and increased radiographic bone gain.
PRP permitted a greater reduction in probing pocket depth, gain in attachment level and amount of radio density with respect to the control group.
The combined mechanical/antimicrobial treatment for the chlorhexidine group did not result in any reduction in probing depth and but only limited reduction of bleeding scores.
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Size reduction in probes and interconnected cannulae is a promising solution to improve temporal and spatial resolution.
To enable unsupervised cluster analysis, we performed unbiased reduction in probe number from around 60,000 to several thousands of probes.
A related decrease in fluorescent labeling was quantified by flow cytometry, resulting in a significant (p < 0.0001) ∼50% reduction in probe-labeled AFA at the cell surface, when ligand addition preceded dye addition.
We observed a wide spectrum of sensitivity, with most CDXG kinases requiring micromolar concentrations of hypothemycin to block labeling by probe 2. Two of the kinases showing the greatest reduction in probe labeling after exposure to hypothemycin, TbGSK3short and TbCLK1, are also the only CDXG kinases whose knockdown by RNAi significantly reduced cell growth.
Significant changes in probing reduction, clinical attachment gain and vertical relative attachment gain suggested that PRP may led to more favourable results compared to HA alone.
It also facilitates reductions in probing depth and bleeding on probing over 12 weeks following scaling and root planning (Johannsen et al. 2009) and inhibits plaque growth (Rodrigues et al. 2010).
In probing for cellular sites of Cr VI) reduction, it was of utmost importance to maintain the normal cellular physiology and growth of the microorganisms.
In fact, our data showed that cotreatment of testosterone and letrozole further attenuated A β-induced reduction in FM probe fluorescent intensity.
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